Carmarthen Veterinary Centre
Farmer Factsheets

 

Post Calving Nutritional Disorders

Fatty Liver Syndrome

o Occurs when DMI falls during late pregnancy, while at the same time energy demand is rising.
o Unless the energy density of the ration is increased to compensate, the cow has to mobilise her own energy reserves.
o As body fat is broken down, triglycerides build up in the liver.

Clinical signs

o Ketosis - early in lactation rather than at peak-yield.
o Concurrent mild conditions that the cow would normally resolve, eg. mastitis, metritis, milk fever, which respond poorly to treatment.

Prevention

o Ensure that the ration supplies adequate energy despite the falling intakes in the 2 weeks before calving.

Ketosis/Acetonaemia

o Usually occurs 4-6 weeks post calving when yield is increasing, but DMI is not increasing to match.
o Clinical signs appear when this negative balance is exacerbated by any other condition that reduces intake, eg. disease or inadequate trough space.

Clinical signs may include:

o Gradual loss of condition, slow decline in yield
o Inappetance
o More sudden weight loss and milk yield drop
o "Pear drops smell" on breath
o Nervous signs: salivation, chewing, licking, in-coordination, aggression.

Prevention

o Minimise energy deficit at calving with good transition cow nutrition.
o Maximise intake post-calving: gradual diet changes, good feed presentation etc.

Displaced Abomasums

o 80% occur in the first month of lactation.

Clinical signs

o Inappentance, especially concentrates.
o Milk drop.
o Decreased rumen movements.

Risk Factors

o Periparturient diseases: RFM, mastitis, metritis and milk fever are all associated with increased incidence of displacements
o Nutrition: increasing concentrate to forage ratio in the diet increases the risk of displacements.
o Position of the abomasum: it is suggested that the change in the position that occurs after calving predisposes to displacement.
o Fibre: feeding diets and forages low in fibre reduces rumen fill and predisposes cattle to displacements.
o Intake: low DMI, ketosis and negative energy balance are all associated with increasing likelihood of abomasal displacements.

Prevention

 

Individual Animal Herd
Manage condition score carefully Careful feeding of dry and transition cows
Monitor for signs of ketosis o Gradually introduce concentrates
Monitor for milk fever o Ensure adequate fibre
Prevent and treat periparturient disease o Maximise DMI post-calving
Monitor rumen fill Consider metabolic profiles
Consider genetic predisposition

 

 


 

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